Document
type: Class

Continuant
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Entity
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Review
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Generically Dependent Continuant
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individual
rights: termsOfUse
date: 2026-08-24T04:54:25
publisher: vivo
type: Document
label: RDF description of miRNAs in nutrition, obesity, and cancer: The biology of miRNAs in metabolic disorders and its relationship with cancer development - http://orbis.uaslp.mx/vivo/individual/scopus7117

miRNAs in nutrition, obesity, and cancer: The biology of miRNAs in metabolic disorders and its relationship with cancer development
has funding vehicle: Consejo Nacional de Ciencia y Tecnología, CONACYT
chemicalscas: MicroRNAs
origen: scopus
type: Information Content Entity
doi: 10.1002/mnfr.201600994
type: Generically Dependent Continuant
most specific type: Document
type: Review
date time value: review date 7118
issue: 1
volume: 62
related by: authorship_45988
freetext keyword: Cancer; Immune cells; Inflammation; miRNAs; Obesity microRNA; animal; complication; genetic epigenesis; human; malnutrition; metabolic disorder; neoplasm; obesity; physiology; tumor microenvironment; Animals; Epigenesis, Genetic; Humans; Malnutrition; Metabolic Diseases; MicroRNAs; Neoplasms; Obesity; Tumor Microenvironment
related by: authorship_45987
estado: published
abstract: Scope: The scope of this review is to explain how metabolic disorders originated by a deficient nutrition can develop into a neoplastic process by the alteration of epigenetic mechanisms like miRNAs. Obesity is a proinflammatory state with a wide impact on health around the world that is associated with neoplastic diseases. Epigenetic mechanisms have a central role in the obesogenic environment, which participates on the development of comorbidities such as cancer. Methods and results: We made an exhaustive review of the most recent reports about metabolic disorders with nutrition and their relationship with miRNAs, and their risk of developing into oncogenic processes. MicroRNAs (miRNAs) act as one of the major epigenetic mechanisms that can affect the metabolic reprogramming of cellular metabolism that plays an important role in the oncogenic process. There is evidence that some foods may contribute to diminishing the risk of cancer as well as epidemiological studies that support the notion that diets high in animal protein and fat promote cancer risk. Therefore, diets high in fruit and vegetables reduce the risk of cancer. One of the principal explanations is that these foods contain bioactive compounds that increase the efficacy of epigenetic mechanisms, which in turn decrease the risk of obesity and its comorbidities. Conclusion: In this review, we show how miRNAs are implicated in several signaling pathways as well as illustrating some bioactive compounds that impact inflammation and cancer development. © 2017 WILEY-VCH Verlag GmbH %26 Co. KGaA, Weinheim
artnum: 1600994
type: Thing
type: Document
e iscopus: 2-s2.0-85026444828
type: Article
related by: authorship_45989
type: Continuant
type: Entity
pmid: 28594107
has publication venue: Molecular Nutrition and Food Research
crisid: 7118
most specific type: Review

Article
type: Class

Molecular Nutrition and Food Research
publication venue for: miRNAs in nutrition, obesity, and cancer: The biology of miRNAs in metabolic disorders and its relationship with cancer development
type: Information Content Entity
type: Continuant
type: Periodical
type: Document
type: Entity
type: Journal
type: Generically Dependent Continuant
type: Thing
type: Collection

Consejo Nacional de Ciencia y Tecnología, CONACYT
type: Relationship
type: BFO_0000020
type: Entity
type: Thing
type: Grant
type: Continuant
funding vehicle for: miRNAs in nutrition, obesity, and cancer: The biology of miRNAs in metabolic disorders and its relationship with cancer development

Information Content Entity
type: Class