Reciprocal interaction between the suprachiasmatic nucleus and the immune system tunes down the inflammatory response to lipopolysaccharide
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Several studies have shown circadian variations in the response of the immune system suggesting a role of the suprachiasmatic nucleus (SCN). Here we show that lipopolysaccharide (LPS) administration in the beginning of the active period induced more severe responses in temperature and cytokines than LPS given in the rest period. Moreover night administered LPS increased SCN basal neuronal activity indicating a direct influence of inflammation on the SCN. Bilateral lesions of the SCN resulted in an increased inflammatory response to LPS demonstrating that an interaction between the SCN and the immune system modulates the intensity of the inflammatory response. © 2014 Elsevier B.V.
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Circadian rhythms; Cytokines; LPS; Temperature response; Wistar rats interleukin 6; lipopolysaccharide; protein c fos; tumor necrosis factor alpha; analysis of variance; animal cell; animal tissue; article; body temperature; brain function; circadian rhythm; controlled study; core temperature; cytokine production; cytokine release; dose response; fever; immune response; immunohistochemistry; inflammation; male; nonhuman; prediction; priority journal; protein blood level; protein expression; rat; suprachiasmatic nucleus; thermoregulation; Circadian rhythms; Cytokines; LPS; Temperature response; Wistar rats; Adjuvants, Immunologic; Animals; Body Temperature; Cell Count; Circadian Rhythm; Cytokines; Dose-Response Relationship, Drug; Immune System; Inflammation; Lipopolysaccharides; Male; Neurons; Proto-Oncogene Proteins c-fos; Rats; Rats, Wistar; Suprachiasmatic Nucleus; Time Factors
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