Involvement of nitric oxide and ATP-sensitive potassium channels in the peripheral antinoceptive action of a tramadol-dexketoprofen combination in the formalin test
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Preclinical Research Systemic coadministration of tramadol and dexketoprofen can produce antinociceptive synergism in animals. There has been only limited evaluation of this drug combination in the peripheral nervous system in terms of the antinociceptive interaction and its mechanisms. The aim of the present study was to evaluate the peripheral antinociceptive interaction between tramadol and dexketoprofen in the formalin test and the involvement of the nitric oxide (NO)-cyclic guanosine monophosphate pathway and ATP-sensitive K channels. Different doses of tramadol or dexketoprofen were administered locally to the formalin-injured mouse paw and the antinociceptive effect evaluated. ED50 values were calculated for both drugs alone and in combination. Coadministration of tramadol and dexketoprofen produced an antinociceptive synergistic interaction during the second phase of the formalin test. Pretreatment with NO antagonists, including l-NG-nitroarginine methyl ester and 1H-[1,2,4]-oxadiazolo-[4,3-a]-quinoxalin-1-one, or the ATP-sensitive K channel antagonist glibenclamide reversed the antinociceptive synergistic effect of the tramadol-dexketoprofen combination, suggesting that NO and ATP-sensitive K channels were involved. © 2014 Wiley Periodicals, Inc.
Preclinical Research Systemic coadministration of tramadol and dexketoprofen can produce antinociceptive synergism in animals. There has been only limited evaluation of this drug combination in the peripheral nervous system in terms of the antinociceptive interaction and its mechanisms. The aim of the present study was to evaluate the peripheral antinociceptive interaction between tramadol and dexketoprofen in the formalin test and the involvement of the nitric oxide (NO)-cyclic guanosine monophosphate pathway and ATP-sensitive K%2b channels. Different doses of tramadol or dexketoprofen were administered locally to the formalin-injured mouse paw and the antinociceptive effect evaluated. ED50 values were calculated for both drugs alone and in combination. Coadministration of tramadol and dexketoprofen produced an antinociceptive synergistic interaction during the second phase of the formalin test. Pretreatment with NO antagonists, including l-NG-nitroarginine methyl ester and 1H-[1,2,4]-oxadiazolo-[4,3-a]-quinoxalin-1-one, or the ATP-sensitive K%2b channel antagonist glibenclamide reversed the antinociceptive synergistic effect of the tramadol-dexketoprofen combination, suggesting that NO and ATP-sensitive K%2b channels were involved. © 2014 Wiley Periodicals, Inc.
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dexketoprofen; formalin test; synergism; tramadol 1h 1,2,4 oxadiazolo[4,3 a]quinoxalin 1 one; adenosine triphosphate sensitive potassium channel; cyclic GMP; dexketoprofen; dimethyl sulfoxide; formaldehyde; glibenclamide; n(g) nitroarginine methyl ester; nitric oxide; tramadol; 1H-(1,2,4)oxadiazolo(4,3-a)quinoxalin-1-one; adenosine triphosphate sensitive potassium channel; analgesic agent; dexketoprofen; glibenclamide; ketoprofen; n(g) nitroarginine methyl ester; nitric oxide; oxadiazole derivative; quinoxaline derivative; tramadol; trometamol; animal experiment; antinociception; Article; controlled study; dose response; drug dose; drug mechanism; drug potentiation; formalin test; licking; male; mouse; nociception; nonhuman; analogs and derivatives; animal; antagonists and inhibitors; drug effects; metabolism; pain measurement; Analgesics; Animals; Dose-Response Relationship, Drug; Drug Synergism; Glyburide; KATP Channels; Ketoprofen; Male; Mice; NG-Nitroarginine Methyl Ester; Nitric Oxide; Oxadiazoles; Pain Measurement; Quinoxalines; Tramadol; Tromethamine
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