Human vascular smooth muscle cells from diabetic patients are resistant to induced apoptosis due to high Bcl-2 expression
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An emerging body of evidence suggests that vascular remodeling in diabetic patients involves a perturbation of the balance between cell proliferation and cell death. Our aim was to study whether arteries and vascular smooth muscle cells (VSMCs) isolated from diabetic patients exhibit resistance to apoptosis induced by several stimuli. Internal mammary arteries (IMAs) were obtained from patients who had undergone coronary artery bypass graft surgery. Arteries from diabetic patients showed increasing levels of Bcl-2 expression in the media layer, measured by immunofluorescence and by Western blotting. Human IMA VSMCs from diabetic patients showed resistance to apoptosis, measured as DNA fragmentation and caspase-3 activation, induced by Creactive protein (CRP) and other stimuli, such as hydrogen peroxide and 7β-hydroxycholesterol. The diabetic cells also exhibited overexpression of Bcl-2. Knockdown of Bcl-2 expression with Bcl-2 siRNA in cells from diabetic patients reversed the resistance to induced apoptosis. Consistent with the above, we found that pretreatment of nondiabetic VSMCs with high glucose abolished the degradation of Bcl-2 induced by CRP. Moreover, cell proliferation was increased in diabetic compared with nondiabetic cells. This differential effect was potentiated by glucose. We conclude that the data provide strong evidence that arterial remodeling in diabetic patients results from a combination of decreased apoptosis and increased proliferation. © 2006 by the American Diabetes Association.
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CRP, C-reactive protein; ERK1/2, extracellular signal-related kinases 1 and 2; IMA, internal mammary artery; TBS-T, Tris-buffered saline with tween; VSMC, vascular smooth muscle cell 7beta hydroxycholesterol; antidiabetic agent; C reactive protein; caspase 3; collagen; DNA fragment; elastin; glucose; hydrogen peroxide; insulin; protein bcl 2; protein bcl xl; small interfering RNA; collagen; elastin; glucose; mannitol; protein bcl 2; adult; aged; apoptosis; article; cell culture; cell isolation; cell proliferation; controlled study; coronary artery bypass graft; diabetes mellitus; dose response; drug effect; gene overexpression; genetic transfection; human; human cell; immunofluorescence; immunofluorescence test; internal mammary artery; isolated artery; priority journal; protein analysis; protein degradation; protein expression; vascular smooth muscle; Western blotting; apoptosis; coronary blood vessel; diabetic angiopathy; female; gene expression regulation; genetics; heart muscle revascularization; male; middle aged; pathology; pathophysiology; physiology; vascular smooth muscle; Aged; Apoptosis; Collagen; Coronary Vessels; Diabetic Angiopathies; Elastin; Female; Gene Expression Regulation; Glucose; Humans; Internal Mammary-Coronary Artery Anastomosis; Male; Mannitol; Middle Aged; Muscle, Smooth, Vascular; Proto-Oncogene Proteins c-bcl-2
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