Mexican HIV-1 Protease Sequence Diversity
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Protease is one of three enzymes encoded within HIV%27s pol gene, responsible for the cleavage of viral Gag-Pol polypeptide into mature viral proteins and a target of current anti-retroviral therapy. Protease diversity analysis in Latin America has been lacking in spite of extensive studies of protease-inhibitor resistance mutations. We studied the diversity of 777 Mexican protease sequences and found that all were subtype B except one (CRF02_AG). Phylogenetic analysis suggested the existence of six different clades with geospecific contributions. Thirty-three percent of sites were conserved, 25%25 had conservative substitutions, and 41%25 exhibited physicochemical changes. The most conserved regions surrounded the active site, most of the flap domain, and a region between the 60%27s loop and C-terminal triad. A single sequence exhibited an active site mutation (T26S). Variable sites were mapped to a crystallographic structure, providing further insight into the distribution and functional relevance of variable sites among Mexican isolates. © Copyright 2020, Mary Ann Liebert, Inc.
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Protease is one of three enzymes encoded within HIV's pol gene, responsible for the cleavage of viral Gag-Pol polypeptide into mature viral proteins and a target of current anti-retroviral therapy. Protease diversity analysis in Latin America has been lacking in spite of extensive studies of protease-inhibitor resistance mutations. We studied the diversity of 777 Mexican protease sequences and found that all were subtype B except one (CRF02_AG). Phylogenetic analysis suggested the existence of six different clades with geospecific contributions. Thirty-three percent of sites were conserved, 25%25 had conservative substitutions, and 41%25 exhibited physicochemical changes. The most conserved regions surrounded the active site, most of the flap domain, and a region between the 60's loop and C-terminal triad. A single sequence exhibited an active site mutation (T26S). Variable sites were mapped to a crystallographic structure, providing further insight into the distribution and functional relevance of variable sites among Mexican isolates. © Copyright 2020, Mary Ann Liebert, Inc.
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anti-retroviral mutations; genetic diversity; Mexico; molecular modelling; protease Gag protein; Human immunodeficiency virus proteinase; saquinavir; antiretrovirus agent; Human immunodeficiency virus proteinase; p16 protease, Human immunodeficiency virus 1; amino acid sequence; antiviral resistance; Article; carboxy terminal sequence; cladistics; consensus sequence; dimerization; enzyme active site; genetic variability; human; Human immunodeficiency virus 1; Jalisco; Mexican; microbial diversity; Morelos; nonhuman; nucleotide sequence; Nuevo Leon; phylogenetic tree; physical chemistry; priority journal; protein domain; Puebla (state); Veracruz (state); virus isolation; virus mutation; drug effect; enzymology; genetic variation; genetics; Human immunodeficiency virus 1; Human immunodeficiency virus infection; Mexico; missense mutation; molecular model; phylogeny; sequence analysis; virology; Anti-Retroviral Agents; Drug Resistance, Viral; Genetic Variation; HIV Infections; HIV Protease; HIV-1; Humans; Mexico; Models, Molecular; Mutation, Missense; Phylogeny; Sequence Analysis, Protein
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