Kaempferol inhibits apoptosis in vascular smooth muscle induced by a component of oxidized LDL
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Antioxidants such as flavonoids afford protection against oxysterols-induced toxicity. We have investigated the effect of kaempferol and rutin, active components of red wine, in the apoptosis induced by 7β-hydroxycholesterol in rat vascular smooth muscle cells. 7β-Hydroxycholesterol induced apoptosis in vascular smooth muscle which include BcL-xL degradation, caspase-3 activation and DNA fragmentation. The apoptosis induced by 7β-hydroxycholesterol was prevented by pretreatment with kaempferol (10-30 μM), but not with rutin. Interestingly preincubation with the estrogen receptor α antagonist ICI 182,780 (1 μM) prior to kaempferol partially reverted the antiapoptotic effect of this flavonoid on caspase-3 activation and DNA fragmentation induced by 7β-hydroxycholesterol. In conclusion, the flavonoid kaempferol, unlike rutin, diminished the apoptosis induced by a component of oxidized low-density lipproteins (oxLDL). This effect was partially mediated by the estrogen receptor α. © 2005 Elsevier B.V. All rights reserved.
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7β-hydroxycholesterol; Apoptosis; Kaempferol; Vascular smooth muscle 7beta hydroxycholesterol; caspase 3; DNA fragment; estrogen receptor alpha; flavonoid; fulvestrant; kaempferol; oxidized low density lipoprotein; protein bcl xl; receptor blocking agent; rutoside; animal cell; animal tissue; apoptosis; article; controlled study; drug effect; drug mechanism; enzyme activation; male; nonhuman; priority journal; protein degradation; rat; red wine; smooth muscle fiber; vascular smooth muscle; Animals; Antioxidants; Aorta, Thoracic; Apoptosis; bcl-X Protein; Cells, Cultured; Estrogen Receptor alpha; Hydroxycholesterols; Kaempferols; Lipoproteins, LDL; Male; Muscle, Smooth, Vascular; Rats; Rats, Wistar; Rutin
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